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Hence, the inactivation or loss of GPX4 due to genetic inhibition or inhibitors or GSH depletion could result in L-OOH accumulation, triggering ferroptotic cell death (Yang et al., 2014)

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Critically, the trial also ran monotherapy arms: −14.9% with semaglutide alone and −11.5% with cagrilintide alone (Garvey et al., 2025)