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N-acetylcysteine depresses contractile function and inhibits fatigue of diaphragm in vitro
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Collectively, these mechanisms encompassing PI3K/Akt activation, modulation of Sirt1-Foxo1, reduction of oxidative stress, inhibition of apoptosis, and cholinergic receptor-mediated anti-inflammatory effects, provide a robust biological basis for curcumin’s neuroprotective and cytoprotective effects in PD (Nebrisi, 2021)
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